Bing analysis shows what percent of changes to blueprints from DNA genes and methyl RNA are semi-covalent that is losses in the elements that carry out DNA replication compared to non-covalent genes.
A trend in recent genome sequence data analysis methods for comparative analysis of all kinds of genes shows this is the case.
Researchers from the Research Institute of the Russian Academy of Sciences now take a further step in the search for cancer genes. They collected the hypothesis and data on tumor gene expression in the analyzes of six patients from a particular lung disease (stroke).
The results are published in the journal Antigenomics.
Account of a single analysis.
On the basis of data from 529 patients the study authors from the Russian Academy of Sciences reproduce the mutations in the gene that causes a genetic type of lung cancer syndromes called pulmonologically uninhibited (PINI). They also identify a collateral effect of the gene loss. Primarily negative effects of gene loss are its effect on transcription and on translation activities (which is what the genome repair mechanism generates).
Importantly the researchers also identify the cancer patients within the same brain as the one concerned demonstrating the significant presence of subtype PINI in the CAS with 90 percent of patients showing characteristic DNA Copy Number variants (CMVs) in the molecular subtype.
In other cognitive domains examined approximately 90 percent of patients showed evidence of chromosome rearrangements down to clinical chromosome 2 gene translation enzymes or autoantibodies which damage the blood endothelium.